| ¿µ¹® | corticosteroid | ÇÑ±Û | ÄÚ¸£Æ¼ÄÚ½ºÅ×·ÎÀ̵å |
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| ¼³¸í | ÄáÆÏÀ§¿¡ ÀÖ´Â »ï°¢»ÔÀÇ ÇüŸ¦ Áö´Ñ ±â°üÀÎ ºÎ½ÅÀÇ °ÑºÎºÐÀÎ °ÑÁú¿¡¼ ¸¸µé¾îÁö´Â ¹°Áú·Î È£¸£¸óÀÇ ÀÏÁ¾. ÄÚ¸£Æ¼°í½ºÅ×·ÎÀ̵å´Â ±× ¿ªÇÒ¿¡ µû¶ó¼ ´ÙÀ½°ú °°Àº 3°¡Áö·Î ³ª´©¾îÁø´Ù. 1.±Û·çÄÚÄÚ¸£Æ¼ÄÚÀ̵å(glucocorticoid)-Áö¹æÁú, ´çÁú, ´Ü¹éÁúÀÇ ´ë»ç¿¡ °ü°èÇϴ ȣ¸£¸ó. Áö¹æÁú°ú ´Ü¹éÁúÀ» ºÐÇØÇØ¼ ´çÁúÀ» ¸¸µå´Â ¿ªÇÒÀ» ÇÏ°í ¶Ç Ã¼³»¿¡ ÀúÀåµÇ¾î ÀÖ´Â ´çÁúÀ» Ç÷¾×¼ÓÀ¸·Î ¹èÃâÇÏ´Â ¿ªÇÒÀ» Çϴ ȣ¸£¸ó. ºÎ½Å°ÑÁú¿¡¼ ¸¸µé¾îÁö´Â ´ç·ùÄÚ¸£Æ¼ÄÚÀ̵å´Â ´ëºÎºÐ ÄÚ¸£Æ¼¼Ö(cortisol)À̶ó´Â ¹°ÁúÀÌ´Ù. 2.±¤¹°ÄÚ¸£Æ¼ÄÚÀ̵å(mineralocorticoid)-ü³»ÀÇ ÀüÇØÁú°ú ¼öºÐÀÇ Á¶Àý¿¡ °ü¿©Çϴ ȣ¸£¸ó. ÄáÆÏ¿¡ ÀÛ¿ëÇÏ¿© ¼öºÐ°ú ³ªÆ®·ýÀÇ ¹è¼³À» ¾ïÁ¦ÇÏ¿© ¼öºÐ, ³ªÆ®·ýÀÇ Ã¼³» ¾çÀ» Áõ°¡½ÃŰ´Â ¿ªÇÒÀ» ÇÑ´Ù. 3.¿¡½ºÆ®·Î°Õ, ¾Èµå·Î°Õ-¼ºÈ£¸£¸óÀ¸·Î ¼ºÀÇ ºÐȳª ¿©·¯ °¡Áö »ý½Ä¿¡ °ü·ÃµÈ ¿ªÇÒÀ» ÇÑ´Ù. ¾Ë¾ÆµÎ¾î¾ß ÇÒ °ÍÀº ºÎ½Å°ÑÁú¿¡¼ »ý»êµÈ À§ÀÇ 3°¡Áö È£¸£¸óÀº Á¤È®È÷ ±â´ÉÀÇ ±¸ºÐÀÌ ÀÖ´Â °ÍÀÌ ¾Æ´Ï°í ´Ù¸¥ È£¸£¸óÀÇ ÀÛ¿ëµµ °¡Áö°í ÀÖ´Â ¼ö°¡ ¸¹´Ù. Áï ±Û·çÄÚÄÚ¸£Æ¼ÄÚÀ̵嵵 ¾à°£ÀÇ ±¤¹°ÄÚ¸£Æ¼ÄÚÀ̵åÀÇ ¿ªÇÒÀÌ ÀÖ°í, ±¤¹°ÄÚ¸£Æ¼ÄÚÀ̵嵵 ¾à°£ÀÇ ±¤¹°ÄÚ¸£Æ¼ÄÚÀ̵åÀÇ ¿ªÇÒÀÌ ÀÖ´Ù. |
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| ¿µ¹® | immunological reaction | ÇÑ±Û | ¸é¿ª¹ÝÀÀ |
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| ¼³¸í | »ýüÀÇ ¸ö ¾È¿¡¼ »ý±ä ¹°ÁúÀ̳ª ¸ö ¹Û¿¡¼ µé¾î¿Â ¹°ÁúÀÌ »ýü¿Í ´Ù¸¦ ¶§ ÀÚ±â ü³»ÀÇ ÅëÀϼº°ú °³Ã¼ÀÇ »ýÁ¸ À¯Áö ¹× Á¾ÀÇ Á¸¼ÓÀ» À§ÇÏ¿© ±× ¹°ÁúµéÀ» Á¦°ÅÇÏ´Â ÀÏ·ÃÀÇ »ýü ¹ÝÀÀ. ´Ù½Ã ¸»ÇØ B¼¼Æ÷¿¡ ÀÇÇÑ Ç×ü»ý»ê, T¼¼Æ÷¸¦ Áß½ÉÀ¸·Î ÇÏ´Â ¼¼Æ÷¼º ¸é¿ª, ¸é¿ª°ü¿ë, ¸é¿ª±â¾ï µîÀÇ »ýü ³» ¹ÝÀÀÀ» ¸»ÇÑ´Ù. Å«Æ÷½Ä¼¼Æ÷´Â Ç׿øÀ» ó¸®Çؼ ƯÀÌÀûÀÎ Ç׿ø°áÁ¤±â¸¦ °®´Â ºÐÀÚ·Î ¹Ù²ã, Ç׿ø°ú ÁÖ¿äÁ¶Á÷ ÀûÇÕÀ¯ÀüÀÚº¹ÇÕü¸¦ ¼¼Æ÷Ç¥¸é¿¡ Ç¥ÇöÇϸç, T¼¼Æ÷·Î Àü´ÞÇÑ´Ù. ÇÑÆí B¼¼Æ÷´Â Å«Æ÷½Ä¼¼Æ÷ ³»¿¡¼ ó¸®µÈ Ç׿øÀÇ °áÁ¤±â¸¦ ÀνÄÇÏ¿© ´ëÀÀÇϴ ƯÀÌÀûÇ×ü¸¦ »ý»êÇÏ¿© Ç׿øÀ» ó¸®ÇÑ´Ù. |
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| ¿µ¹® | reaction formation | ÇÑ±Û | ¹Ýµ¿Çü¼º, ¹ÝÀÀÇü¼º |
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| ¼³¸í | ¾ï¾Ðº¸´Ù ´õ Àû±ØÀûÀÎ ¹æ¾î¸ÞÄ¿´ÏÁòÀ̸ç, ¹«ÀǽÄÀûÀÎ »ý°¢, ¼Ò¿ø, Ãæµ¿ÀÌ ³Ê¹«³ªµµ ¹Þ¾Æµé¿©Áú ¼ö ¾ø´Â °ÍÀÏ °æ¿ì¿¡ À̿ʹ Á¤¹Ý´ë ¹æÇâÀÇ °ÍÀ» °Á¶ÇÔÀ¸·Î½á ±×·± ¹«ÀǽÄÀûÀÎ °ÍµéÀÌ ÀǽĵÇÁö ¾Ê°Ô ÇÏ´Â °úÁ¤. ¿¹¸¦ µé¸é °¡Àå °¡ÇÐÀûÀÎ ¼º°ÝÀÇ »ç¶÷ÀÌ »ýÃ¼ÇØºÎ ¹Ý´ë·ÐÀÚ°¡ µÇ´Â °æ¿ì¸¦ µé ¼ö ÀÖ´Ù. À̰ÍÀº ¶Ç °¡½¿ ±íÀÌ Àá°ÜÀÖ´Â µÎ·Á¿òÀÌ ÀǽĵǴ °ÍÀ» ÇÇÇϱâ À§Çؼ µÎ·Á¿òÀÇ ´ë»óÀÌ µÇ´Â Çൿ¿¡ °ñ¸ôÇÏ´Â °æ¿ìµµ Æ÷ÇÔÀÌ µÈ´Ù. ¿¹¸¦ µé¸é, ³²ÀÚ¿¡°Ô »óó¹ÞÁö ¾ÊÀ»±î ÇÏ´Â µÎ·Á¿ò¿¡ °¡µæ Âù ¼Ò³à°¡ ÀÌ °°Àº µÎ·Á¿òÀ» ºÎÁ¤ÇÏ·Á´Â ¼ö´ÜÀ¸·Î ³ÀâÇÑ ¼ºÇàÀ§¿¡ °ñ¸ôÇÏ´Â °æ¿ì°¡ ÀÖ´Ù. ¶Ç ÀüóÀÇ Àڳฦ ¹Ì¿öÇÏ´Â °è¸ð°¡ ¿ÀÈ÷·Á Áö³ªÄ¥ Á¤µµ·Î ±× ¾ÆÀ̸¦ ±Í¿©¿öÇÏ´Â ÀÏ µûÀ§ÀÌ´Ù. |
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| ¿µ¹® | complement fixation reaction | ÇÑ±Û | º¸Ã¼°áÇÕ ¹ÝÀÀ, µµ¿òü°áÇÕ¹ÝÀÀ |
|---|---|---|---|
| ¼³¸í | Ç×ü¿ÍÀÇ ¹ÝÀÀ¿¡ ÀÖ¾î¼ º¸Ã¼¿Í °áÇÕÇÏ´Â Ç×ü¸¦ °Ë»çÇÏ´Â ¹æ¹ýÀ¸·Î, ÀÌ ¹ÝÀÀÀº ÃÖÃÊ¿¡ ±âÁöÇ׿ø, ÇǰËÇ÷û ¹× º¸Ã¼¸¦ È¥ÇÕÇÑ´Ù. Á¦2´Ü°è¿¡¼´Â ÀûÇ÷±¸¿Í À̰Ϳ¡ ´ëÀÀÇÏ´Â ¿ëÇ÷¼ÒÀÇ È¥ÇÕ¾×À» °¡ÇÑ´Ù. º» ¹ÝÀÀÈÄ ¿ëÇ÷ÀÌ ÀϾÁö ¾ÊÀ¸¸é º»Ã¼´Â Ç׿øÇ×ü°áÇÕ¹°¿¡ °áÇÕÇÑ °ÍÀÌ µÇ¾î ¾ç¼ºÀÌ µÇÁö¸¸, ¿ëÇ÷ÀÌ ÀÏ¾î³ °æ¿ì º¸Ã¼´Â °áÇÕÇÏÁö ¾Ê¾Æ ¼ÒºñµÇÁö ¾Ê±â ¶§¹®¿¡ À½¼ºÀÌ µÈ´Ù. º» ¹ÝÀÀÀº ±âÁöÇ÷ûÀ» ½á¼ Ç׿ø°ËÃâ¿¡ ÀÀ¿ëÇÒ ¼ö ÀÖÀ¸¸ç, ¸¶ÀÌÄÚÇö󽺸¶, ¸®ÄÉÃ, Ŭ¶ó¹Ìµð¾Æ, ¹ÙÀÌ·¯½º, ¸Åµ¶ µîÀÇ Áø´Ü¿¡ ¾²ÀδÙ. |
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| ¿µ¹® | transfusion reaction | ÇÑ±Û | ¼öÇ÷ºÎÀÛ¿ë, ¼öÇ÷¹ÝÀÀ |
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| ¼³¸í | ¼öÇ÷ÇÏ¿´À» ¶§¿¡ ȯÀÚ¿¡°Ô ÀϾ´Â ¹ÝÀÀ. ¾Ë·¹¸£±â ¹ÝÀÀ°ú ¿ëÇ÷ ¹ÝÀÀÀÌ ÀÖ´Ù. |
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| LR | labeled release; laboratory references; laboratory report; labor room; lactated Ringer [solution]; l... |
|---|---|
| CMO | Corticosteroid Methyl-Oxidase |
| 17-OHCS | 17-OH(Hydroxy)-Corticosteroid |
| CBG | capillary blood gases; coronary bypass graft; corticosteroid-binding globulin; cortisol-binding glob... |
| CBGv | corticosteroid-binding globulin variant |
| 11-OHCS | 11-hydroxy-corticosteroid |
|---|---|
| 17-OHCS | 17-hydroxy-corticosteroid |
| C | Corticosteroid |
| CBG | Corticosteroid Binding Globulin |
| CR | Corticosteroid-resistant |
| corticosteroid | <drug, endocrinology, pharmacology> Any of the steroids elaborated by the adrenal cortex (excluding the sex hormones of adrenal origin) in response to the release of adrenocorticotrophin or adrenocorticotropic hormone (ACTH) by the pituitary gland, to any of the synthetic equivalents of these steroids or to angiotensin II. They are divided, according to their predominant biological activity, into three major groups: glucocorticoids (e.g. Cortisol, cortisone), chiefly influencing carbohydrate, fat and protein metabolism, mineralocorticoids (for example aldosterone), affecting the regulation of electrolyte and water balance and C19 androgens. Some corticosteroids exhibit both types of activity in varying degrees and others exert only one type of effect. The corticosteroids are used clinically for hormonal replacement therapy, for suppression of ACTH secretion by the anterior pituitary, as antineoplastic, antiallergic and anti-inflammatory agents and to suppress the immune response. Synonym: adrenocortical hormone, corticoid. (13 Nov 1997) |
|---|---|
| corticosteroid-binding globulin | <chemical> Chemical name: Transcortins (12 Dec 1998) |
| corticosteroid-binding protein | <chemical> Chemical name: Transcortins (12 Dec 1998) |
| corticosteroid cream | <drug, pharmacology> This is a large group of trade name medications that contain an anti-inflammatory steroid. These cream based medications all contain either hydrocortisone in varying concentrations or a synthetic steroid with much greater potency. This type of medications is useful in the treatment of a large number of inflammatory rashes. Caution: higher potency creams can cause side effects. Examples include hydrocortisone, dexamethasone, flunisolide, fluocinonide, fluprednisolone, Aclovate, Alphatrex, Aristocort, Betatrex, Cortone, Diprolene, Valisone, Halog, Hytone, Kenalog, Lidex, Lotrisone, Synalar, Synemol, Topicort, Vytone, Westcort and Vioform. (27 Sep 1997) |
| corticosteroid-induced acne | The exacerbation of acne after corticosteroid use is a common drug reaction seen in adolescents. Lessening the dose of the corticosteroid will often diminish this effect. (27 Sep 1997) |
| corticosteroid-induced glaucoma | <ophthalmology> Glaucoma caused by a hereditary predisposition in which local instillation of eyedrops containing corticosteroid causes increased intraocular pressure. (05 Mar 2000) |
| corticosteroid-induced striae | <dermatology> High-dose glucocorticoid therapy causes atrophy of the skin and a failure of the normal regenerative process. Purple striae often develop over the individual's trunk or thighs. The colour fades when the drug is stopped, but the dermal atrophy remains. (27 Sep 1997) |
| corticosteroid side-chain-isomerase | <enzyme> Converts 11-deoxycorticosterone to 20-hydroxy-3-oxypregn-4-en-21-al; also acts as an epimerase at c-20 Registry number: EC 5.3.1.21 Synonym: corticosteroid side chain isomerase, ccsci (26 Jun 1999) |
| accelerated reaction | A response occurring in a shorter time than expected; the cutaneous manifestations occurring during the period between the second and tenth day following smallpox vaccination; because it is intermediate between a primary reaction and an immediate reaction, it is regarded as evidence of some degree of resistance. Synonym: vaccinoid reaction. (05 Mar 2000) |
| acid reaction | Any test by which an acid reaction is recognised such as the change of blue litmus paper to red, an excess of hydrogen ions over hydroxide ions in aqueous solution indicated by a pH value less than 7 (at 22°C). Compare: dissociation constant of water. (05 Mar 2000) |
| acute-phase reaction | <immunology, rheumatology> Refers to the changes in synthesis of certain proteins within the serum during an inflammatory response, which provides rapid protection for the host against microorganisms via non-specific defense mechanisms. It consists of fever, an increase in inflammatory humoral factors, and an increased synthesis by hepatocytes of a number of proteins or glycoproteins usually found in the plasma; the reaction is mediated by endogenous pyrogens, the hypothalamus, adrenal hormones, and other factors. (12 Jul 2000) |
| acute situational reaction | An acute emotional reaction related to extreme environmental stress. Synonym: acute situational reaction. (05 Mar 2000) |
| acute stress reaction | A sudden bout of anxiety that is often accompanied by the features of hyperventilation (tingling around mouth and in fingertips, rapid breathing, faintness or fainting). (27 Sep 1997) |
| addition reaction | <chemistry> Any reaction in which two chemicals combine to form a single chemical. (15 Jan 1998) |
| adverse drug reaction reporting systems | Systems developed for collecting reports from government agencies, manufacturers, hospitals, physicians, and other sources on adverse drug reactions. (12 Dec 1998) |
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