| ¿µ¹® | receptor | ÇÑ±Û | ¼ö¿ëü |
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| ¼³¸í | ¼¼Æ÷Áú³» ¶Ç´Â ¼¼Æ÷Ç¥¸é¿¡ Á¸ÀçÇÏ´Â ºÐÀÚ±¸Á¶·Î¼ ƯÀ̹°Áú°ú ¼±ÅÃÀûÀ¸·Î °áÇÕÇÏ¸ç °áÇÕ¿¡ ÀÇÇØ ƯÀÌÇÑ »ý¸®Àû ÀÛ¿ëÀ» ³ªÅ¸³½´Ù. ÆéƼµåÈ£¸£¸ó, ½Å°æÀü´Þ¹°Áú, Ç׿ø, º¸Ã¼, ¸é¿ª±Û·ÎºÒ¸°¿¡ ´ëÇÑ ¼¼Æ÷Ç¥¸é ¼ö¿ëü¿Í ½ºÅ×·ÎÀ̵忡 ´ëÇÑ ¼¼Æ÷Áú³» ¼ö¿ëü°¡ ÀÖ´Ù. |
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| ¿µ¹® | cholinergic | ÇÑ±Û | Äݸ°(ÀÛµ¿)¼º |
|---|---|---|---|
| ¼³¸í | 1.½Å°æ¼¼Æ÷°¡ ´Ù¸¥ ½Å°æ¼¼Æ÷·Î Á¤º¸¸¦ Àü´ÞÇÒ °æ¿ì¿¡´Â ´ëºÎºÐ ÈÇй°ÁúÀ» ºÐºñÇÏ¿© ±×°ÍÀ» ¸Å°³·Î ÇÏ¿© ´Ù¸¥ ½Å°æ¿¡ Á¤º¸¸¦ Àü´ÞÇÑ´Ù. ÀÌ·± ¹°ÁúÀ» ½Å°æÀü´Þ¹°Áú¶ó°í ÇÑ´Ù. Äݸ°¼ºÀ̶õ ½Å°æÀü´Þ¹°Áú·Î acetylcholineÀ» »ç¿ëÇÏ´Â ½Å°æÀ» À̸£´Â ¸»ÀÌ´Ù. Áï ºÎ±³°¨½Å°æÀ» À̸£´Â ¸»·Î ¾²ÀδÙ. 2. Acetylcholine°ú À¯»çÇÑ ¾à¸®Àû ÀÛ¿ëÀ» ÇÏ´Â. |
||
| ER | efficiency ratio; epigastric region; ejection rate; electroresection; emergency room; endoplasmic re... |
|---|---|
| RAR | rapidly adapting receptor; rat insulin receptor; retinoic acid receptor; right arm reclining; right ... |
| CR | calculation rate; calculus removed; calorie-restricted; cardiac rehabilitation; cardiac resuscitatio... |
| CRL | cell repository line; Certified Record Librarian; complement receptor location; complement receptor ... |
| DR | degeneration reaction; delivery room; deoxyribose; diabetic retinopathy; diagnostic radiology; digit... |
| CBF | Cholinergic basal forebrain |
|---|---|
| HCNP | Hippocampal cholinergic neurostimulating peptide |
| mAChR | Muscarinic cholinergic receptors |
| NANC | Non-adrenergic non-cholinergic |
| CRLR | Calcitonin Receptor-Like Receptor |
| cholinergic receptors | Chemical sites in effector cells or at synapses through which acetylcholine exerts its action. (05 Mar 2000) |
|---|
| nicotinic cholinergic receptor | A class of receptors responsive to acetylcholine that also are activated by nicotine; ganglionic (including the adrenal medulla) and neuromuscular receptors. Two classes exist: nicotinic-neuronal and nicotinic-muscular. (05 Mar 2000) |
|---|---|
| receptors, cholinergic | Cell surface proteins that bind acetylcholine with high affinity and trigger intracellular changes influencing the behaviour of cells. Cholinergic receptors are divided into two major classes, muscarinic and nicotinic, based originally on their affinity for nicotine and muscarine. Each group is further subdivided based on pharmacology, location, mode of action, and/or molecular biology. (12 Dec 1998) |
| cholinergic | <pharmacology> Resembling acetylcholine in pharmacological action, stimulated by or releasing acetylcholine or a related compound. (15 Jan 1998) |
| cholinergic agent | An agent that mimics the action of the parasympathetic nervous system (e.g., methacholine). (05 Mar 2000) |
| cholinergic agents | Any drug used for its actions on cholinergic systems. Included here are agonists and antagonists, drugs that affect the life cycle of acetylcholine, and drugs that affect the survival of cholinergic neurons. The term cholinergic agents is sometimes still used in the narrower sense of muscarinic agonist, although most modern texts discourage that usage. (12 Dec 1998) |
| cholinergic agonists | Drugs that bind to and activate cholinergic receptors. (12 Dec 1998) |
| cholinergic antagonists | Drugs that bind to but do not activate cholinergic receptors, thereby blocking the actions of acetylcholine or cholinergic agonists. (12 Dec 1998) |
| cholinergic blockade | Inhibition by a drug of nerve impulse transmission at autonomic ganglionic synapses (ganglionic blockade), at postganglionic parasympathetic effector cells (e.g., by atropine), and at myoneural junctions (myoneural blockade), the inhibition of a cholinergic agent. (05 Mar 2000) |
| cholinergic fibres | Nerve fibres liberating acetylcholine at the synapse after an impulse. (12 Dec 1998) |
| cholinergic medication | <pharmacology> Medications which stimulate cholinergic receptors (for example bethanechol). (15 Jan 1998) |
| cholinergic neuron | <physiology> Neurons in which actylcholine is the neurotransmitter. (15 Jan 1998) |
| cholinergic parasympathomimetic agent | <pharmacology> A chemical substance that causes the release of choline (acetylcholine) from parasympathetic nerve endings. (15 Jan 1998) |
| cholinergic system | <physiology> The system of nerve cells that uses acetylcholine as its neurotransmitter, nerve cells in the cholinergic system are damaged in the brains of Alzheimer patients. (15 Jan 1998) |
| cholinergic urticaria | A form of physical or non-allergic urticaria initiated by heat (e.g., hot baths, physical exercise, pyrexia, exposure to sun or to a warm room) or by excitement; the rather distinctive lesions consist of pruritic areas 1 to 2 mm in diameter surrounded by bright red macules. Synonym: heat urticaria. (05 Mar 2000) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| cholinergic receptor |
A site in a nerve synapse or effector cell that responds to the effect of acetylcholine.
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