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KMLE °Ë»ö
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ÀÇÇпë¾î »çÀü
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"central plasticity hypothesis"¿¡ ´ëÇÑ ´ëÇÑÀÇÇù ÀÇÇпë¾î ¼¼ºÎ °Ë»ö °á°úÀÔ´Ï´Ù
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
plasticity
1. Çü¼º·Â, ¼Ò¼º 2. ¼ºÇü·Â
hypothesis
°¡¼³
lattice hypothesis
°ÝÀÚ°¡¼³
null hypothesis
¿µ°¡¼³, ±Í¹«°¡¼³
unitarian hypothesis
Ç×üµ¿Àϰ¡¼³
central
Áß½É-, ÁßÃß-
central amaurosis
ÁßÃß¼ºÈæ¾Ï½Ã
central aphasia
ÁßÃß¾ð¾î»ó½ÇÁõ, ÁßÃ߽ǾîÁõ
central artery
Á߽ɵ¿¸Æ
central ataxia
ÁßÃß½ÇÁ¶
central attachment
Á߽ɺÎÂø
central auditory processing disorder
ÁßÃßû°¢Ã³¸®Àå¾Ö
central callus
Á߽ɾֹú»À, Á߽ɰ¡°ñ
central canal
Á߽ɰü
central cataract
Á߽ɹ鳻Àå
´ÙÀ½
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
6
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
hypothesis
°¡¼³
null hypothesis
±Í¹«°¡¼³, ¿µ°¡¼³
central
Áß½É-, ÁßÃß-
central vein cannulation
Áß½ÉÁ¤¸Æ°ü»ðÀÔ¼ú
central venous pressure
Áß½ÉÁ¤¸Æ¾Ð
central nervous system
ÁßÃ߽Űæ°è, ÁßÃ߽Űæ°èÅë
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
1
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
central plasticity hypothesis
ÁßÃßÀ¯¿¬¼º°¡¼³
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
plasticity
Çü¼º¼º
blending hypothesis
À¶ÇÕÀ¯Àü¼³
countercurrent hypothesis
¿ª·ù°¡¼³
drift hypothesis
À̵¿°¡¼³
dual recognition hypothesis
Ç׿øÀÌÁßÀÎÁö°¡¼³
estrogen window hypothesis
¿¡½ºÆ®·Î°Õâ°¡¼³
expectation hypothesis
¿¹Ãø°¡¼³
hypothesis
°¡¼³
hypoxia-selectivity hypothesis
Àú»ê¼ÒÁõ¼±Åõµ°¡¼³
lattice hypothesis
°ÝÀÚ°¡¼³
lipid hypothesis
ÁöÁú°¡¼³
metabolic hypothesis
´ë»ç°¡¼³
molecular hypothesis
ºÐÀÚ°¡¼³
monoclonal hypothesis
´ÜÀÏŬ·Ð°¡¼³
null hypothesis
±Í¹«°¡¼³
´ÙÀ½
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
Glucostatic hypothesis, of appetite regulation
Ç×´ç±â¼³(ù÷ÓØÐñæò), ½Ä¿åÁ¶Àý(ãÝé¯ðàï½)
Lyon s hypothesis
¶óÀ̿°¡¼³.
Starling s hypothesis
½ºÅ¸¾Ë¸µ°¡¼³.
hypothesis, lattice
°ÝÀÚ¼³
receptor hypothesis
¼ö¿ëü°¡¼³
CPU (central processing unit)
Áß¾Ó Ã³¸® ÀåÄ¡
anterolateral central arteries anterolateral thalamostriate
¾Õ°¡ÂÊÁ߽ɵ¿¸Æ ¾Õ°¡ÂʽûóÁÙ¹«
anteromedial central arteries
¾Õ¾ÈÂÊÁ߽ɵ¿¸Æ
anteromedial central branches
¾Õ¾ÈÂÊÁ߽ɰ¡Áö
artery of central sulcus
Á߽ɰí¶ûµ¿¸Æ
glial cell of central nervous system
ÁßÃ߾Ʊ³¼¼Æ÷
gray central
Áß½Éȸ»öÁú(ñéãýüéßäòõ).
harversian canal central canal
Á߽ɰü
hearing loss, central
ÁßÃß¼º û·Â¼Ò½Ç, ÁßÃß¼º ³Ã»
posterolateral central arteries
µÚ°¡ÂÊÁ߽ɵ¿¸Æ
´ÙÀ½
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
neural, plasticity
atherosclerosis,monoclonal hypothesis
´ÜÀÏŬ·Ð¼º°¡¼³(Ó¤ìé¡Ê£àã)
atherosclerosis,reaction to injury hypothesis
¼Õ»ó¿¡ ´ëÇÑ ¹ÝÀÀ±âÀü
atomic hypothesis
¿øÀÚ°¡¼³(ê«íÊ£æò).
blending hypothesis
À¶ÇÕÀ¯Àü¼³(¡ë¶îîæò).
countercurrent hypothesis
¿ª·ù°¡¼³ (¡Ê£æò).
cyclol hypothesis
»çÀÌŬ·Ñ ´ÜÀ§°¡¼³(¡Ó¤êÈÊ£æò).
dopamine hypothesis
µµÆÄ¹Î °¡¼³
drift hypothesis
À̵¿°¡¼³
dual recognition hypothesis
Ç׿øÀÌÁßÀÎÁö°¡¼³
emphysema,protease-antiprotease hypothesis
´Ü¹é-Ç״ܹéºÐÇØÈ¿¼Ò °¡¼³
estrogen window hypothesis
¿¡½ºÆ®·ÎÁ¨ ±¸°£°¡¼³
expectation hypothesis
¿¹Ãø°¡¼³(çãö´Ê£àã)
fractional kill hypothesis
hypothesis, lattice
°ÝÀÚ¼³
´ÙÀ½
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
ÅëÇÕ°Ë»ö ¿Ï·á
´ÙÀ½