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"alpha antitrypsin deficiency panniculitis"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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¿µ¹® iron deficiency anemia ÇÑ±Û Ã¶°áÇ̺óÇ÷
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¿µ¹® alpha particles ÇÑ±Û ¾ËÆÄÀÔÀÚ
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  ¿øÀÚ¿¡ Á¤»óº¸´Ù °ú´Ù, °ú¼ÒÇÑ Áß¼ºÀÚ³ª ¾çÀÚ¸¦ °¡Áö°í Àִ °æ¿ì¿¡ ¿øÀÚ°¡ ¾ÈÁ¤µÇ±â À§Çؼ­ ºØ±«°¡ »ý±ä´Ù. ÀÌ·± ºØ±«¿¡´Â ´ÙÀ½°ú °°Àº 3°¡Áö ¹æ¹ýÀÌ ÀÖ´Ù. ¾ËÆÄ£­ºØ±«´Â ¾ËÆÄÀÔÀÚ¸¦ ¹æÃâÇÏ¿© ¾ÈÁ¤µÇ´Â ¹ý, Áï ¿øÀÚ¹øÈ£´Â 2¾¿ °¨¼ÒÇϰí Áú·®¼ö´Â 4¾¿ °¨¼ÒÇÑ´Ù. ¾ËÆÄÀÔÀÚ¶õ ¾ËÆÄºØ±«¿¡ ÀÇÇØ¼­ »ý±â´Â Çï·ýÀÇ ÇÙ°ú °°Àº ÀÔÀÚ¸¦ ¸»ÇÑ´Ù.
  
  
¿µ¹® alpha-fetoprotein ÇÑ±Û ¾ËÆÄžƴܹé
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  Å¾ÆÀÇ °£Á¶Á÷À̳ª ¼ÒÈ­±â°ü¿¡¼­ ¸¸µé¾îÁö´Â ´Ü¹éÁú. Å¾Ʊ⳪ ÀÓ»êºÎ¿¡¼­ Á¤»óÀûÀ¸·Î Á¸ÀçÇÑ´Ù. ±×·¯³ª °£¾Ï¼¼Æ÷³ª »ý½Ä¼¼Æ÷¿¡ °ü·ÃµÈ Á¾¾ç¿¡¼­µµ ¸¸µé¾îÁ® ÀÌ·± Áúº´ÀÌ Á¸ÀçÇÒ ¶§¿¡´Â ÀÓ»êºÎ³ª Å¾ư¡ ¾Æ´Ï´õ¶óµµ Á¶Á÷À̳ª Ç÷¾×¿¡ ³ªÅ¸³ª°Ô µÈ´Ù. ÀÓ»óÀûÀ¸·Î´Â °£¾ÏÀ̳ª »ý½Ä¼¼Æ÷ Á¾¾çÀÇ ¹ß°ß°ú Ä¡·áÈ¿°ú ÆÇÁ¤¿¡ ÀÌ¿ëµÇ°í, ¶Ç ÀӽŠ16~18ÁÖ¿¡ »ê¸ðÀÇ Ç÷¾×¿¡¼­ À̴ܹéÁúÀÇ ¾çÀ» ÃøÁ¤ÇÏ¿© Å¾ÆÀÇ ºñÁ¤»óÀûÀΠ¹ß´ÞÀ» ¹ß°ßÇÒ ¼ö ÀÖ´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • antitrypsin
    Çׯ®¸³½Å
  • alpha1-antitrypsin
    ¾ËÆÄ1-Çׯ®¸³½Å
  • factitious panniculitis
    ÀΰøÁö¹æÃþ¿°
  • lipoatrophic panniculitis
    Áö¹æÀ§ÃàÁö¹æÃþ¿°
  • liquefying panniculitis
    ¾×È­Áö¹æÃþ¿°
  • lobular panniculitis
    ¼Ò¿±Áö¹æÃþ¿°
  • nodular panniculitis
    °áÀýÁö¹æÃþ¿°
  • panniculitis
    Áö¹æÃþ¿°
  • subcutaneous panniculitis-like T-cell lymphoma
    ÇÇÇÏÁö¹æÃþ¿°À¯»çT¼¼Æ÷¸²ÇÁÁ¾
  • systemic nodular panniculitis
    Àü½Å°áÀýÁö¹æÃþ¿°
  • alpha emitter
    ¾ËÆÄ¹æÃâü
  • alpha error
    ¾ËÆÄ¿À·ù, Á¦1Çü¿À·ù
  • alpha granule
    ¾ËÆÄ°ú¸³
  • alpha hemolysis
    ¾ËÆÄ¿ëÇ÷
  • alpha motor neuron
    ¾ËÆÄ¿îµ¿½Å°æ¼¼Æ÷
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 6 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • panniculitis
    Áö¹æÃþ¿°
  • iron deficiency anemia
    ö°áÇ̺óÇ÷
  • deficiency
    °áÇÌ(Áõ)
  • alpha-interferon
    ¾ËÆÄÀÎÅÍÆä·Ð
  • alpha-adrenergics
    ¾ËÆÄ¾Æµå·¹³¯¸°¼º¾à
  • alpha-fetoprotein
    ¾ËÆÄžƴܹé
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • antitrypsin
    Çׯ®¸³½Å
  • factitial panniculitis
    ÀΰøÁö¹æÃþ¿°
  • lipoatrophic panniculitis
    Áö¹æÀ§Ã༺Áö¹æÃþ¿°
  • liquefying panniculitis
    ¾×È­Áö¹æÃþ¿°
  • lobular panniculitis
    ¼Ò¿±»óÁö¹æÃþ¿°
  • nodular panniculitis
    °áÀýÁö¹æÃþ¿°
  • panniculitis
    Áö¹æÃþ¿°
  • systemic nodular panniculitis
    Àü½Å°áÀýÁö¹æÃþ¿°
  • alpha-chymotrypsin
    ¾ËÆÄÄ«ÀÌ¸ðÆ®¸³½Å
  • alpha-fetoprotein
    ¾ËÆÄžƴܹé
  • alpha-interferon
    ¾ËÆÄÀÎÅÍÆä·Ð
  • alpha-lipoproteinemia
    ¾ËÆÄÁö¹æ´Ü¹éÇ÷Áõ
  • alpha emitter
    ¾ËÆÄ¹æÃâ±â
  • alpha error
    Á¦ÀÏÁ¾¿À·ù
  • alpha granule
    ¾ËÆÄ°ú¸³
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • antitrypsin deficiency
    Çׯ®¸³½Å°áÇÌ
  • galactosidase, alpha-galactosidase a, deficiency
    #NAME?
  • Llipoatrophic panniculitis
    Áö¹æ À§Ã༺ Áö¹æÃþ¿°
  • alpha1-antitrypsin
    ¾ËÆÄ-Çׯ®¸³½Å
  • alpha1-antitrypsin
    ¾ËÆÄ1-Çׯ®¸³½Å
  • antitrypsin
    Çׯ®¸³½Å.
  • antitrypsin test
    Çׯ®¸³½Å½ÃÇè.
  • poststeroid panniculitis
    ½ºÅ×·ÎÀ̵åÅõ¿©ÈÄ Áö¹æÃþ¿°
  • relapsing febrile nodular nonsuppurative panniculitis
    Àç¹ß¼º ¹ß¿­°áÀý¼º ºñÈ­³ó¼º Áö¹æÁú¿°(¡­Û¡æðÌ¿ï½àõÞªûùÒÛàõò·Û¸òõæú)
  • relapsing febrile nodular panniculitis
    Àç¹ß¼º¹ß¿­¼Ò°áÀý¼ºÁö¹æÃþ¿°(¡­Û¡æðá³Ì¿ï½àõò·Û¸õùæú)
  • relapsing panniculitis ; Weber Christian disease
    Àç¹ß¼º(ÇÇÇÏ)Áö¹æÃþ¿° ; ¿þ¹öÅ©¸®½ºÂùº´.
  • Acquiered immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª °áÇÌÁõÈıº
  • Fluorine deficiency
    ºÒ¼Ò°áÇÌÁõ(Ý×áÈÌÀù¹ñø)
  • Folate deficiency
    ¿±»ê°áÇÌÁõ(ç¨ß«ÌÀù¹ñø)
  • Iodine deficiency
    ¿äµå °áÇÌÁõ(ÌÀù¹ñø)
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • alpha antitrypsin deficiency panniculitis
    ¾ËÆÄ Çׯ®¸³½Å°áÇÌÁö¹æÃþ¿°
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • antitrypsin deficiency
    Çׯ®¸³½Å°áÇÌ
  • alpha1-antitrypsin
    ¾ËÆÄ-Çׯ®¸³½Å
  • alpha1-antitrypsin
    ¾ËÆÄ1-Çׯ®¸³½Å
  • antitrypsin
    Çׯ®¸³½Å.
  • antitrypsin test
    Çׯ®¸³½Å½ÃÇè.
  • galactosidase, alpha-galactosidase a, deficiency
    #NAME?
  • mannosidase, alpha-mannosidase, deficiency
    #NAME?
  • cold panniculitis
    ÇÑ·© Áö¹æÃþ¿°(ò·Û¸öµæú)
  • connective tissue panniculitis
    °áÇÕÁ¶Á÷ Áö¹æÃþ¿°
  • eosinophilic panniculitis
    È£»ê±¸¼º Áö¹æÃþ¿°
  • factitial panniculitis
    Àΰø Áö¹æÃþ¿°
  • factitial panniculitis
    ÀÎÀ§Áö¹æÃæ¿°
  • febrile nodular panniculitis
    ¿­¼º °áÀý¼º Áö¹æÃþ¿°
  • histiocytic cytophagic panniculitis
    Á¶Á÷±¸ ½Ä¼¼Æ÷¼º Áö¹æÃþ¿°
  • histiocytic panniculitis
    Á¶Á÷±¸¼º Áö¹æÃþ¿°
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • Alpha cell [Glucagon cell]
    ¾ËÆÄ¼¼Æ÷ [±Û·çÄ«°ï¼¼Æ÷]
    [¿¾ ¿ë¾î] ¾ËÆÄ¼¼Æ÷
  • Deficiency (Nanismus)
    °áÇÌ(³­ÀåÀÌÁõ)
    [¿¾ ¿ë¾î] °áÇÌ
  • Organogenetic deficiency
    ±â°ü¹ß»ý°áÇÌ
    [¿¾ ¿ë¾î] ±â°ü¹ß»ý°áÇÌ
  • Functional deficiency
    ±â´É°áÇÌ
    [¿¾ ¿ë¾î] ±â´ÉÀû°áÇÌ
  • Pituitary hormone deficiency (Pituitary dwarfism)
    ³úÇϼöüȣ¸£¸ó°áÇÌ (³úÇϼöü³­ÀåÀÌ)
    [¿¾ ¿ë¾î] ³úÇϼöüȣ¸£¸ó°áÇÌ (³úÇϼöü³­ÀåÀÌ)
  • Reaction deficiency
    ¹ÝÀÀ°áÇÌ
    [¿¾ ¿ë¾î] ¹ÝÀÀ°áÇÌ
  • Vitamin deficiency (Fetal osteodystrophy)
    ºñŸ¹Î°áÇÌ(žƻÀ¿µ¾çÀå¾Ö)
    [¿¾ ¿ë¾î] ºñŸ¹Î°áÇÌ(žƻÀ¿µ¾çÀå¾Ö)
  • Cellular deficiency (Acallosal cerebrum)
    ¼¼Æ÷°áÇÌ (³úµéº¸°á¿©³ú)
    [¿¾ ¿ë¾î] ¼¼Æ÷°áÇÌ
  • Intracellular deficiency (Albinism)
    ¼¼Æ÷¼Ó°áÇÌ (¹é»öÁõ)
    [¿¾ ¿ë¾î] ¼¼Æ÷³»°áÇÌ
  • Chromosomal deficiency
    ¿°»öü°áÇÌ
    [¿¾ ¿ë¾î] ¿°»öü°á½Ç
  • Stimulation deficiency
    ÀڱذáÇÌ
    [¿¾ ¿ë¾î] ÀڱذáÇÌ
  • Sensory deficiency
    °¨°¢°áÇÌ
    [¿¾ ¿ë¾î] °¨°¢°áÇÌ
  • Thyroid hormone deficiency (Cretinism)
    °©»ó»ùÈ£¸£¸ó°áÇÌ (°©»ó»ù³­ÀåÀÌ)
    [¿¾ ¿ë¾î] °©»ó¼±È£¸£¸ó°áÇÌ (°©»ó»ù³­ÀåÀÌ)
  • Deficiency
    °áÇÌ
    [¿¾ ¿ë¾î] °áÇÌ
  • Deficiency (Monstrous tumor)
    °áÇÌ (±«¹°Á¾)
    [¿¾ ¿ë¾î] °áÇÌ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • antitrypsin
    Ç×(ù÷)Æ®¸³½Å
  • alpha adrenergic receptor
    ¾ËÆÄ¾Æµå·¹³ª¸°ÀÛµ¿¼º(íÂÔÑàõ) ¼ö¿ëü(áôé»ô÷)
  • alpha amanitin
    ¾ËÆÄ¾Æ¸¶´Ïƾ
  • alpha amino acid
    ¾ËÆÄ¾Æ¹Ì³ë»ê(ß«)
  • alpha blocker
    ¾ËÆÄÂ÷´ÜÁ¦(ó´Ó¨ð¥)
  • alpha bungarotoxin
    ¾ËÆÄºÐ°¡·ÎÅå½Å
  • alpha fraction
    ¾ËÆÄºÐȹ(ÝÂüñ)
  • alpha orientation
    ¾ËÆÄ¿À¸®¿£Å×À̼Ç
  • alpha receptor
    ¾ËÆÄ¼ö¿ëü(áôé»ô÷)
  • alpha tocopherol
    ¾ËÆÄÅäÄÚÆä·Ñ
  • conditioned vitamin deficiency
    Á¶°ÇºÎ(ðÉËìݾ) ºñŸ¹Î°áÇË(ÌÀù¹)
  • deficiency
    °áÇÌ(ÌÀù¹)
  • deficiency disease
    °áÇÌ Áúȯ(ÌÀù¹òðü´)
  • deficiency mutant
    "°áÇÌ º¯ÀÌü(ÌÀù¹Ü¨ì¶ô÷), (ÔÒ) auxotroph"
  • dietary deficiency
    ½ÄÀ̰áÇÌ(ç½å×ÌÀù¹)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 10 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • panniculitis
    Áö¹æÃþ¿°
  • alpha
    ¾ËÆÄ
  • alpha fetoprotein
    ¾ËÆÄžƴܹé
  • alpha ray
    ¾ËÆÄ¼±
  • deficiency
    °áÇÌÁõ
  • deficiency anemia
    °áÇ̼ººóÇ÷
  • iron deficiency anemia
    ö°áÇ̼ººóÇ÷
  • mineral deficiency
    ¹«±âÁú°áÇÌ(Áõ)
  • nutritional deficiency disease
    ¿µ¾ç°áÇÌÁõ
  • vitamin deficiency
    ºñŸ¹Î°áÇÌ(Áõ)
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
AAD acute agitated delirium; alloxazine adenine dinucleotide; alpha-1-antitrypsin deficiency; American A...
NLP no light perception; nodular liquefying panniculitis; normal light perception; normal luteal phase
MD Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major...
AAT Aachen Aphasia Test; academic aptitude test; alanine aminotransferase; alkylating agent therapy; alp...
a1AT alpha-1-antitrypsin
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
alpha1ATD Alpha-1-antitrypsin deficiency
ATD 1-antitrypsin deficiency
AATD Alpha1-antitrypsin deficiency
CHP Cytophagic histiocytic panniculitis
alpha 1-AT Alpha 1 antitrypsin
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • cold panniculitis
    ÇÑ·© Áö¹æÃþ¿°
  • eosinophilic panniculitis
    È£»ê±¸¼º Áö¹æÃþ¿°
  • factitial panniculitis
    Àΰø Áö¹æÃþ¿°
  • A alpha
    A ¾ËÆÄ, A ¾ËÆÄ ½Å°æ ¼¶À¯
  • A alpha primary afferent
    A ¾ËÆÄ ÀÏÂ÷ ±¸½É ½Å°æ
    ±Ù¹æÃß ³»ÀÇ ±Ù ¼¶À¯¿¡ Á¸ÀçÇÏ´Â °¨°¢½Å°æÀÇ Çϳª·Î ¥°a °¨°¢ ½Å°æÀ̶ó°íµµ ÇÑ´Ù. Á÷°æÀº 22§­, Àüµµ ¼Óµµ´Â 120§½ÀÌ´Ù.
  • A-alpha mechanoreceptor
    A ¾ËÆÄ ±â°è ¼ö¿ë±â
    ±Ù ¹æÃß¿Í °ñÁö °Ç ±â°ü¿¡ Á¸ÀçÇÑ´Ù.
  • alpha
    ¥á
    chain
  • alpha 1-adrenergic blocker
    ¾ËÆÄ 1-¾Æµå·¹³¯¸°¼º Â÷´Ü
  • alpha 2 agonist clonidine
    ¾ËÆÄ 2 ÀÛµ¿ Ŭ·Î´Ïµò
  • alpha 2 binding
    ¾ËÆÄ 2 °áÇÕ
  • alpha adrenergic antagonist
    ¾ËÆÄ ¾Æµå·¹³¯¸° ±æÇ×Á¦
  • alpha amylose
    ¾ËÆÄ-¾Æ¹Ð·Î½º
    °ÅÀÇ ¾Æ¹Ð·Î½ºÀÎ ÀüºÐÀÇ ÁÖ¼ººÐÀ¸·Î¼­, ±Û·çÄÚ½º°¡ a-1,4 °áÇÕÀ¸·Î Á÷¼â»ó °áÇÕÇÑ °Í.
  • alpha efferent motor neuron
    ¾ËÆÄ ¿ø½É ¿îµ¿ ´º¿ì·±, ¾ËÆÄ ¿ø½É¼º ¿îµ¿ ´º·±
  • alpha fiber
    ¾ËÆÄ ¼¶À¯, ¾ËÆÄ ½Å°æ¼¶À¯
  • alpha hemolysis
    ¾ËÆÄ ¿ëÇ÷
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
alpha-1 antitrypsin deficiency <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues.
The lack of this protein leads to damage of various organs, but mainly to the lung and liver.
symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant
(12 Dec 1998)
alpha-antitrypsin deficiency <enzyme> A specific enzyme (alpha 1 antitrypsinase) that when absent genetically can result in panacinar emphysema (lung disease) and liver disease.
There is no specific treatment for this condition other than supportive care for the liver and lung complications.
Medications such as alpha-1proteinase inhibitor is given regularly to these patients.
Incidence: approximately 1 in 10,000.
(02 Jan 1998)
deficiency, alpha-1 antitrypsin An inherited disease with little or no production of an important protein, alpha-1 antitrypsin. The lack of this protein leads to damage of various organs, mainly the lung and liver. The disease may become apparent at a very early age or in adulthood, as shortness of breath or liver-related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant
(12 Dec 1998)
antitrypsin deficiency Deficiency of a1-antitrypsin, a glycoprotein of the postalbumin region of human serum. Many forms are known which may be moderate (40 to 60% of normal activity) or severe (less than 10% of normal), all autosomal dominant; the severe form is often associated with familial emphysema or hepatic cirrhosis.
(05 Mar 2000)
alpha-1 antitrypsin <chemical> Plasma glycoprotein member of the serpin superfamily which inhibits trypsin, neutrophil elastase, and other proteolytic enzymes. Commonly referred to as alpha 1-proteinase inhibitor (a1pi), it exists in over 30 different biochemical variant forms known collectively as the pi (protease inhibitor) system.
Hereditary deficiency is associated with pulmonary emphysema.
See: alpha-1 antitrypsin deficiency
Pharmacological action: serine proteinase inhibitors, trypsin inhibitors.
(12 Dec 1998)
panniculitis <pathology> An inflammatory reaction of the subcutaneous fat, which may involve the connective tissue septa between the fat lobes, the septa lobules and vessels or the fat lobules, characterised by the development of single or multiple cutaneous nodules.
(18 Nov 1997)
panniculitis, lupus erythematosus A type of lupus erythematosus characterised by deep dermal or subcutaneous nodules, most often on the head, face, or upper arms. It is generally chronic and occurs most often in women between the ages of 20 and 45.
(12 Dec 1998)
panniculitis, nodular nonsuppurative A form of panniculitis characterised by recurrent episodes of fever accompanied by the eruption of single or multiple erythematous subcutaneous nodules on the lower extremities. They normally resolve, but tend to leave depressions in the skin. The condition is most often seen in women, alone or in association with other disorders.
(12 Dec 1998)
panniculitis, peritoneal Condition of the peritoneum, most commonly of the mesentery, but also of the omentum, characterised by tissue thickening, alteration of fat cells, infiltration of lipid-laden macrophages, and fibrosis.
(12 Dec 1998)
poststeroid panniculitis Subcutaneous nodules developing in children within a month after withdrawal of corticosteroids given to treat the nephrotic syndrome or rheumatic fever; microscopically identical to subcutaneous fat necrosis of the newborn, the condition resolves spontaneously or with steroid readministration.
Relapsing febrile nodular nonsuppurative panniculitis, nodular fat necrosis of a variety of possible causes.
Synonym: Christian's disease, nodular nonsuppurative panniculitis, Weber-Christian disease.
Subacute migratory panniculitis, non-scarring plaques of changing configuration on the lateral aspect of one or both legs, of many months duration.
Synonym: erythema nodosum migrans.
(05 Mar 2000)
nodular nonsuppurative panniculitis relapsing febrile nodular nonsuppurative panniculitis
a1-antitrypsin A glycoprotein that is the major protease inhibitor of human serum, is synthesised in the liver, and is genetically polymorphic due to the presence of over 20 alleles; individuals appropriately homozygous are deficient in a1-trypsin and are predisposed to pulmonary emphysema and juvenile hepatic cirrhosis because of alterations in the amino acid and sialic acid components of the glycoprotein. A1-Antitrypsin also inhibits thrombin.
Synonym: a1-trypsin inhibitor, human a1-proteinase inhibitor.
(05 Mar 2000)
antitrypsin <protein> This is a glycoprotein produced in the liver which is the major antiprotease in the blood, serving mainly to inhibit leukocyte elastase.
It is a single-chain molecule, has 394 amino acids and a molecular weight of 51,000.
Hereditary antitrypsin deficiency is one of the most common genetic disorders among males of European descent. Alpha-1-antitrypsin deficiency results in leukocyte elastase building up and breaking down the lining of the lung, which results in a severe form of emphysema (oxidizing agents in cigarette smoke are also known to inactivate antitrypsin, thus causing the high rates of emphysema among long-term smokers).
Because over 20,000 people are affected by this deficiency, large amounts of this antiprotease are needed. To meet the demand, researchers are genetically engineering sheep that produce the antiprotease in their milk.
(13 Jan 1998)
alpha-1-proteinase deficiency Absence of a serum proteinase inhibitor that may cause nodular non-suppurative panniculitis.
(05 Mar 2000)
alpha, alpha-phosphotrehalase <enzyme> Forms glucose plus glucose-6-phosphate
Registry number: EC 3.2.1.93
Synonym: trehalose-6-phosphate hydrolase, phospho-alpha(1,1)glucosidase, trea gene product, trec gene product
(26 Jun 1999)
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