| ¿µ¹® | receptor | ÇÑ±Û | ¼ö¿ëü |
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| ¿µ¹® | progesterone | ÇÑ±Û | ÇÁ·Î°Ô½ºÅ×·Ð, Ȳüȣ¸£¸ó |
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| ¼³¸í | 21°³ÀÇ Åº¼Ò¸¦ °¡Áö´Â ½ºÅ×·ÎÀ̵å·Î¼ ÁÖ·Î ³¼ÒÀÇ È²Ã¼ ¹× Źݿ¡¼ ¸¸µé¾îÁö³ª, ±× ¿Ü °íȯÀ̳ª ºÎ½Å°ÑÁú¿¡¼µµ ¹Ì·® ¸¸µé¾îÁø´Ù. ÀÌ È£¸£¸óÀº Àڱÿ¡ ÀÛ¿ëÇÏ¿© ¼öÁ¤¶õ(Á¤ÀÚ¿Í ³ÀÚ°¡ ¸¸³ª¼ »ý±ä žÆÀÇ Àü±¸Ã¼)À» Âø»ó½Ã۱â À§ÇÑ Áغñ·Î Àڱ󻸷À» ¼ºÀå½ÃŰ°í ¹ß´Þ½Ã۴µ¥ °ü¿©Çϰí ÀÓ½ÅÀÌ µÇ¸é, Ȳü´Â ´ë°³ 1°³¿ù°£ ÇÁ·Î°Ô½ºÅ×·ÐÀ» ºÐºñÇÏ¿© ÀÓ½ÅÀ» À¯Áö½Ã۸ç, ±× ÀÌÈÄ¿¡´Â Źݿ¡¼ ºÐºñÇÑ´Ù. ¼öÁ¤ÀÌ ÀϾÁö ¾ÊÀ¸¸é, Ȳü´Â ´õÀÌ»ó ÇÁ·Î°Ô½ºÅ×·ÐÀ» ºÐºñÇÏÁö ¾ÊÀ¸¹Ç·Î, Àڱ󻸷ÀÌ Å»¶ôµÇ¾î ¿ù°æÀÌ ½ÃÀ۵ȴÙ. ±× ¿Ü À¯¹æ¿¡ ÀÛ¿ëÇÏ¿© Á¥ÀÇ »ý»êÀº Áõ°¡½Ã۰í, ºÐºñ¸¦ ¾ïÁ¦ÇÏ´Â ÀÛ¿ëµµ ÀÖ´Ù. |
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| ¿µ¹® | test | ÇÑ±Û | °Ë»ç |
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| ¼³¸í | ¾î¶² ´Ù¸¥ ¹°ÁúÀ» °ËÃâ, ÃøÁ¤, »ý¼ºÇϱâ À§ÇÑ Æ¯Á¤ÇÑ ÈÇйÝÀÀÀ» ÀÏÀ¸Å°´Âµ¥ »ç¿ëµÇ´Â ¹æ¹ý. |
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| ¿µ¹® | scratch test | ÇÑ±Û | ³Àý¹ý |
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| ¼³¸í | ÇǺθ¦ ³¯Ä«·Î¿î ¹Ù´Ã·Î ±Ü¾î ÇǺÎÀÇ ¹ÝÀÀÀ» º¸´Â °Ë»ç·Î ÇǺΠ°ú¹Î¹ÝÀÀÀ̳ª ¾Ë·¹¸£±â¸¦ ¾Ë¾Æº¸±â À§ÇÑ °Ë»çÀÌ´Ù. ¹Ù´Ã³¡¿¡ Ç׿øÀ» ¹¯Èù µÚ, ÇǺιØÀ» ±Ü¾î ¹ÝÀÀÀ» ¾Ë¾Æº»´Ù. À̶§ Ç׿øÀÌ ¾Æ´Ñ ´ëÁ¶¹°Áú(¿¹¸¦ µé¾î º¸ÅëÀÇ ¹°)À» ¹¯Èù ¹Ù´ÃÀ» °°ÀÌ ¹ÝÀÀÇÏ¿© ÇǺ馱âÁõ(dermographism) ´ÜÁö ¹Ù´ÃÀÇ ±ÜÈû¸¸À¸·Î ¾Ë·¹¸£±â °°Àº ¹ÝÀÀÀ» ÀÏÀ¸Å°´Â Çö»ó°ú °¨º°ÇØ¾ß ÇÑ´Ù. ³Ã», û·Â¼Ò½Ç(hearing loss) û°¢ÀÌ ÀúÇÏ ¶Ç´Â »ó½ÇµÈ »óÅÂ. ¿øÀΰú Á¤µµ´Â ¿©·¯ °¡ÁöÀε¥, ³Ã»Àº ±× Á¤µµ°¡ °¡Àå ½ÉÇÑ »óÅÂÀÌ´Ù. û°¢ÀÇ Àüµµ°æ·Î¿¡ Àå¾Ö°¡ ÀÖÀ» ¶§ ³Ã»ÀÌ ÀϾ°í, ±× º´ÅͰ¡ ¿ÜÀ̵µ³ª ÁßÀÌ¿¡ ÀÖ´Â °ÍÀ» ÀüÀ½³Ã», ³»ÀÌ¿¡ ÀÖ´Â °ÍÀ» °¨À½ ³Ã»À̶ó ÇÏ¿© ±¸ºÐÇÑ´Ù. ¶Ç º´ÅÍÀÇ ÀÚ¸®¸¦ ¸í½ÃÇÏ¿© ÁßÀ̼º ³Ã»À̳ª ¹Ì·Î¼º ³Ã» µîÀ¸·Î ¼¼ºÐÇϱ⵵ ÇÑ´Ù. |
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| ¿µ¹® | stool guaiac test | ÇÑ±Û | ´ëº¯ ±¸¾ÆÀÌ¾Ç °Ë»ç |
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| ¼³¸í | ´ëº¯³»¿¡ ÀÖÀ» ¼ö ÀÖ´Â ÀáÇ÷(´«¿¡ º¸ÀÌÁö ¾Ê´Â ÃâÇ÷)À¯¹«¸¦ °Ë»çÇÏ´Â ¹æ¹ýÀ¸·Î, Ç÷±¸³»ÀÇ heme peroxidase¿¡ ÀÇÇØ guaiacÀÌ »êȵǴ ¹ÝÀÀÀ» ÀÌ¿ëÇÏ¿© ÃøÁ¤ÇÑ´Ù. ¹æ¹ýÀº 3Àϰ£¿¡ °ÉÃÄ ÇÑ º¯¿¡¼ 2±ºµ¥¾¿ äÃëÇÏ¿© °Ë»çÇÑ´Ù. À§¾ç¼º ¹ÝÀÀ(°ÅÁþÀ¸·Î Ç÷¾×ÀÌ ÀÖ´Ù°í ³ªÅ¸³ª´Â ¹ÝÀÀ)Àº ½Ä¹° °ú»êÈÈ¿¼Ò¸¦ ÇÔÀ¯Çϰí ÀÖ´Â È«´ç¹«ÀÇ ¼·Ã볪 Ç÷±¸ ¼ººÐÀ» ÇÔÀ¯Çϰí ÀÖ´Â °í±â ¼·Ãë µî¿¡¼ ³ªÅ¸³¯ ¼ö ÀÖÀ¸¸ç, À§À½¼º ¹ÝÀÀ(½ÇÁ¦·Î Ç÷¾×Àº ÀÖÁö¸¸, Ç÷¾×ÀÌ ¾ø´Ù°í ³ªÅ¸³ª´Â ¹ÝÀÀ)Àº ȯ¿ø·ÂÀ» °¡Áö°í ÀÖ´Â ºñŸ¹Î CÀÇ º¹¿ë½Ã ³ªÅ¸³¯ ¼ö ÀÖ´Ù. ƯÈ÷ À§¾ç¼º ¹ÝÀÀÀÌ ¸Å¿ì ÈçÇÏ´Ù. |
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| HCG, hCG | Human Chorionic Gonadotropin; »ç¶÷À¶¸ð¼º¼º¼±ÀÚ±ØÈ£¸£¸ó 1. Placental Glycoprotein Hormone &nbs... |
|---|---|
| PCT | peripheral carcinoid tumor; plasma clotting time; plasmacrit test; plasmacytoma; polychlorinated tri... |
| PAT | Pain Apperception Test; paroxysmal atrial tachycardia; patient; phenylaminotetrazole; physical abili... |
| PGR | progesterone receptor; psychogalvanic response |
| PgR | progesterone receptor |
| PR | Progesterone Receptor |
|---|---|
| PgR | Progesterone receptor |
| CPR | chicken progesterone receptor |
| E test | Epsilometer test |
| 17 OHP | 17 OH Progesterone |
| progesterone receptor assay | The progesterone receptor test (PgR assay) checks the tumour for its hormone status. (16 Dec 1997) |
|---|---|
| receptors, progesterone | Specific proteins found in or on cells of progesterone target tissues that specifically combine with progesterone. The cytosol progesterone-receptor complex then associates with the nucleic acids to initiate protein synthesis. There are two kinds of progesterone receptors, a and b. Both are induced by oestrogen and have short half-lives. (12 Dec 1998) |
| progesterone | <biochemistry, gynaecology> Produced in the corpus luteum, as an antagonist of oestrogens. Promotes proliferation of uterine mucosa and the implantation of the blastocyst, prevents further follicular development. (18 Nov 1997) |
| progesterone-11HS-horseradish peroxidase | <chemical> Used as a tracer in an immunoassay for progesterone Synonym: p-11hs-hrp, progesterone-11hs-hrp (26 Jun 1999) |
| progesterone 20-keto-reductase | <enzyme> Rat liver microsome enzyme requires NADPH to form the 20beta alcohol, also forms 20alpha oh using NADH Registry number: EC 1.1.1.- Synonym: progesterone 20-reductase, progesterone 20-beta reductase (26 Jun 1999) |
| progesterone-3-(O-carboxymethyl oxime)-horseradish peroxidase | <chemical> Used as a tracer in an immunoassay for progesterone Synonym: progesterone-3-(o-carboxymethyl oxime)-hrp, p-3cmo-hrp (26 Jun 1999) |
| progesterone 5 alpha-reductase | <enzyme> Do not confuse this with progesterone reductase which is a 3 beta-hydroxysteroid dehydrogenase Registry number: EC 1.3.1.30 (26 Jun 1999) |
| progesterone 5 beta-reductase | <enzyme> A stereospecific enzyme; forms 5 beta-pregnane-3,20-dione from progesterone; see also progesterone 5 alpha-reductase Registry number: EC 1.3.1.- (26 Jun 1999) |
| progesterone-6-hydroxylase | <enzyme> Cytochrome p-450 2c3 with valine at position 364 which confers 6beta-hydroxylase activity; see also record for cyp2c3 gene product Registry number: EC 1.14.99.- Synonym: progesterone 6beta-hydroxylase, cytochrome p450 2c3v (26 Jun 1999) |
| progesterone-binding globulin | A glycoprotein migrating as alpha 1-globulin, molecular weight 70,000 to 120,000. The protein, which is present in increased amounts in the plasma during pregnancy, binds mainly progesterone, with other steroids including testosterone competing weakly. (12 Dec 1998) |
| progesterone reductase | <enzyme> An enzyme that catalyses the reduction of a 3 beta-hydroxy-delta(5)-steroid to 3-oxo-delta(4)-steroid in the presence of NAD. It converts pregnenolone to progesterone and dehydroepiandrosterone to androstenedione.45. Chemical name: 3 beta-Hydroxy-delta(5)-steroid:NAD+ 3-oxidoreductase Registry number: EC 1.1.1.145 (12 Dec 1998) |
| progesterone unit | The progestational activity of 1 mg of unit of progestational activity; standard preparation of pure progesterone. See: Clauberg test, Corner-Allen unit. Synonym: corpus luteum hormone unit, unit of luteinizing activity. (05 Mar 2000) |
| hormone, progesterone | A female hormone, progesterone is the principal progestational hormone. Progestational hormones prepare the uterus (the womb) to receive and sustain the fertilised egg. (12 Dec 1998) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| amino acid receptor | <biochemistry> Ligand gated ion channels with specific receptors for amino acid transmitters. An extended protein superfamily that also includes subunits of the nicotinic acetylcholine receptor. (18 Nov 1997) |
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