| 영문 | muscle cell(=muscle fiber) | 한글 | 근육세포 |
|---|---|---|---|
| 설명 | 근육은 수의근(의식에 의해서 조절이 가능한 근육: 예를 들어 다리, 팔, 얼굴근육 등)과 불수의근(의식과 무관하게 조절하지 않아도 움직이는 근육: 예를 들어 심장근, 소화기관에 분포하는 근육 등)으로 나누어지며, 근육세포의 모양은 다음과 같다(수의근, 불수의근 외에 심장에 있는 심장근은 불수의근에 해당하지만 그 모양은 수의근과 같아 따로 분류한다).  |
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| 영문 | glycogen | 한글 | 글리코겐, 당원 |
|---|---|---|---|
| 설명 | 동물에서 탄수화물의 저장형태로 주로 간에 대부분이 저장되어 있고 근육에 조금 있다. 포도당으로 분해되어 이용된다. |
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| 영문 | striated muscle | 한글 | 가로무늬근 |
|---|---|---|---|
| 설명 | 표면에 가로줄무늬가 보이는 근육. 뜻대로 움직일 수 있으므로 수의근이라고도 불린다. 인체의 가로무늬근의 대부분은 골격근이며, 얼굴의 피부를 움직이는 표정근, 혀나 후두를 움직이는 근육도 가로무늬근이다. 예를 들어 팔을 구부릴 때는 많은 근육의 복잡한 협조가 필요하여 전체적인 움직임을 통제하는 기구가 있다. 또 자세의 균형을 잡을 때 등 많은 운동을 무의식적-반사적으로 조절하는 기구도 있다. 심장근은 가로무늬근이지만 불수의근의 성질을 가졌다. |
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| 영문 | skeletal muscle | 한글 | 골격근 |
|---|---|---|---|
| 설명 | 골격에 붙어 그 운동을 관장하는 근육계. 골격근-평활근-심장근 등 세 개 근조직의 하나. 굵기 10~100μ, 길이 5~12cm의 가늘고 긴 근육섬유의 집합체이며, 가로무늬가 있고, 수의운동을 한다. 한 개의 골격근은 다수의 근섬유와 결합조직으로 구성되고 각기 특유한 형태를 지닌다. 근육의 양끝은 가늘며 그 부분을 근육머리라고 한다. 근육머리는 힘줄로 이행하며 힘줄은 뼈막에 붙는데, 때로는 뼈막을 뚥고 뼈에 부착되어 있다. 근육의 중앙부는 굵고 두꺼우며 이부분을 근복이라 한다. 근육머리는 다시 두갈래근-세갈래근-네갈래근으로 나뉜다. 근육의 운동 자체는 항상 근섬유의 방향에 따르는 수축운동뿐이다. 그러나 골격근이 뼈에 붙은 위치에 따라 뼈대에 대한 여러 가지 운동을 하게 된다. 운동하는 형태로 골격근을 분류하면 폄근-굽힘근-내전근-외전근-회외근-회내근-올림근 등이 있다. 굽히고 폄-내외전-회내외의 운동은 관절축을 중심으로 행한다. 같은 골격에 대하여 펌근육과 굽힘근육이 각기 반대운동을 할 경우에는 양 근육을 서로 대항근이라 하고, 공동운동을 하는 경우에는 공동근이라 한다. |
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| 영문 | muscle | 한글 | 근육 |
|---|---|---|---|
| 설명 | 의식의 조절여부에 따라 수의근(의식에 의해서 조절이 가능한 근육: 예를 들어 다리, 팔, 얼굴근육 등)과 불수의근(의식과 무관하게 조절하지 않아도 움직이는 근육: 예를 들어 심장근, 소화기관에 분포하는 근육 등)으로 나누어질 수 있으며, 또한 심장근이 따로 존재한다. |
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| MR | Maddox rods; magnetic resistance; magnetic resonance; mandibular reflex; mannose-resistant; may repe... |
|---|---|
| GFFS | glycogen and fat-free solid |
| GSD | genetically significant dose; Gerstmann-Straussler disease; glutathione synthetase deficiency; glyco... |
| GSD-0 | glycogen storage disease-zero |
| GT1-GT10 | glycogen storage disease, types 1 to 10 |
| GP | Glycogen Phosphorylase |
|---|---|
| GSK-3 beta | I)/glycogen synthase kinase-3 beta |
| Gly | Glycogen |
| GSD | Glycogen Storage Disease |
| GSDII | Glycogen Storage Disease type II |
| glycogen phosphorylase | <enzyme> Enzyme that catalyses the sequential removal of glycosyl residues from glycogen to yield one glucose-1-phosphate per reaction. Its activity is controlled by phosphorylation (by phosphorylase kinase). (21 Jun 2000) |
|---|---|
| muscle phosphorylase deficiency | Type V glycogen storage disease, affecting muscle, caused by deficiency of muscle phosphorylase. (05 Mar 2000) |
| brancher glycogen storage disease | Type of glycogen storage disease, due to deficiency of amylo-1,4-1,6-transglucosidase (brancher enzyme). Synonym: brancher deficiency glycogenosis, debrancher deficiency. (05 Mar 2000) |
| glycogen | <biochemistry> Branched polymer of D glucose (mostly _(1-4) linked, but some _(1-6) at branch points). Size range very variable, up to 10exp5 glucose units. Major short term storage polymer of animal cells and is particularly abundant in the liver and to a lesser extent in muscle. In the electron microscope glycogen has a characteristic asterisk or star appearance. (18 Nov 1997) |
| glycogen debranching enzyme system | 1,4-alpha-d-glucan-1,4-alpha-d-glucan 4-alpha-d-glucosyltransferase/dextrin 6 alpha-d-glucanohydrolase. An enzyme system having both 4-alpha-glucanotransferase (ec 2.4.1.25) and amylo-1,6-glucosidase (ec 3.2.1.33) activities. As a transferase it transfers a segment of a 1,4-alpha-d-glucan to a new 4-position in an acceptor, which may be glucose or another 1,4-alpha-d-glucan. As a glucosidase it catalyses the endohydrolysis of 1,6-alpha-d-glucoside linkages at points of branching in chains of 1,4-linked alpha-d-glucose residues. Amylo-1,6-glucosidase activity is deficient in glycogen storage disease type III. (12 Dec 1998) |
| glycogen granule | Glycogen occurring in cells as beta granule's which average about 300 A |
| glycogen storage disease | <hepatology> A group of inherited metabolic disorders involving the enzymes responsible for the synthesis and degradation of glycogen. In some patients, prominent liver involvement is presented. In others, more generalised storage of glycogen occurs, sometimes with prominent cardiac involvement. Synonym: glycogenosis (12 Sep 2002) |
| glycogen storage disease type I | <disease> An autosomal recessive disease in which gene expression of glucose-6-phosphatase is absent, resulting in hypoglycaemia due to lack of glucose production. Accumulation of glycogen in liver and kidney leads to organomegaly, particularly massive hepatomegaly. Increased concentrations of lactic acid and hyperlipidemia appear in the plasma. Clinical gout often appears in early childhood. Inheritance: autosomal recessive. (12 Dec 1998) |
| glycogen storage disease type II | <disease> Glycogenosis due to alpha-1,4-glucosidase (acid maltase) deficiency. It affects muscle, heart, and other organs. (12 Dec 1998) |
| glycogen storage disease type III | <disease> An autosomal recessive metabolic disorder due to deficient expression of amylo-1,6-glucosidase (one part of the glycogen debranching enzyme system). The clinical course of the disease is similar to that of glycogen storage disease type I, but milder. Massive hepatomegaly, which is present in young children, diminishes and occasionally disappears with age. Levels of glycogen with short outer branches are elevated in muscle, liver, and erythrocytes. Six subgroups have been identified, with subgroups type IIIa and type IIIb being the most prevalent. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type IV | <disease> An autosomal recessive metabolic disorder due to a deficiency in expression of branching enzyme (alpha-1,4-glucan-6-alpha-glucosyltransferase), resulting in an accumulation of abnormal glycogen with long outer branches. Clinical features are muscle hypotonia and cirrhosis. Death from liver disease usually occurs before age 2. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type V | <disease> Glycogenosis due to muscle phosphorylase deficiency. Characterised by painful cramps following sustained exercise. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type VI | <disease> A hepatic glycogen storage disease in which there is an apparent deficiency of hepatic phosphorylase activity. However, studies have not been able to distinguish between phosphorylase deficiency and phosphorylase kinase deficiency in patients with hepatic glycogenosis. (12 Dec 1998) |
| glycogen storage disease type VII | <disease> An autosomal recessive muscle glycogen storage disease in which there is deficient expression of muscle phosphofructokinase activity, resulting in increased concentrations of glucose-6-phosphate and fructose-6-phosphate and low concentrations of fructose-1,6-diphosphate in muscle tissue. Glycogen storage in muscle is increased, perhaps due to activation of glycogen synthase by accumulated glucose-6-phosphate. It has been proposed that shunting of glucose-6-phosphate and fructose-6-phosphate into the pentose phosphate pathway may result in increased synthesis of purines and pyrimidines, causing hyperuricaemia and gout. Erythrocytes from patients may show decreased phosphofructokinase activity and 2,3-diphosphoglycerate deficiency. Exercise intolerance is present and severe congenital muscular dystrophy has been reported. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type VIII | <disease> An x-linked recessive hepatic glycogen storage disease resulting from lack of expression of phosphorylase-b-kinase activity. Symptoms are relatively mild; hepatomegaly, increased liver glycogen, and decreased leukocyte phosphorylase are present. Liver shrinkage occurs in response to glucagon. Inheritance: X-linked recessive (12 Dec 1998) |
Synonyms : Glycogen Phosphorylase a, Muscle Form, Glycogen Phosphorylase b, Muscle Form, Myophosphorylase a and b
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|