| ¿µ¹® | repression | ÇÑ±Û | ¾ï¾Ð |
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| ¼³¸í | ¹«ÀǽÄÀûÀ̰í À§ÇùÀûÀÎ Ãæµ¿, °¨Á¤, ¼Ò¿ø, ȯ»ó, ±â¾ï µîÀÌ ÀǽĵÇÁö ¸øÇϵµ·Ï ÇÏ´Â ¹æ¾î±âÁ¦. °¡Àå °£´ÜÇÑ ¿¹·Î ±¥Á¾½Ã°èÀÇ ¼Ò¸®¸¦ ¾ÆÄ§¿¡ ¡®¸øµé¾ú´Ù¡¯´Â °ÍÀÌ´Ù. ±ÍÂúÀº °úÁ¦´Â ¡®Àؾú´Ù¡¯´Â °ÍÀÌ´Ù. À̰ÍÀº ÀØ°í ½ÍÀº ±â¾ïÀ̳ª ¿ë³³µÇÁö ¾Ê´Â ¿å±¸¸¦ ÀǽÄÀûÀ¸·Î ÀØÀ¸·Á ÇÏ´Â ¾ïÁ¦(suppression)°ú´Â ±¸º°µÇ´Â ¹«ÀǽÄÀûÀÎ Çö»óÀ¸·Î ¾ï¾ÐÀÇ ¹æ¾î±âÀüÀÌ ÀÛ¿ëÇÏ´Â »ç½Ç¿¡ ´ëÇØ¼ ÀÚ½ÅÀº ÀǽÄÀûÀ¸·Î´Â Á¤¸» ¸ð¸£°í ÀÖ´Â »óŰ¡ µÈ´Ù. |
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| ¿µ¹® | Repression | ÇÑ±Û | ¾ïÁ¦ |
|---|---|---|---|
| ¼³¸í | À¯ÀüÇп¡¼´Â, ¾ïÁ¦ÀÎÀÚ(repressor)¿¡ ÀÇÇØ À¯ÀüÀÚº¹Á¦(replication)°¡ ¾ïÁ¦µÇ´Â °Í. |
||
| RAGE | Receptor for Advanced Glycation Endproduct |
|---|---|
| CCR | Carbon catabolite repression |
| NCR | Nitrogen catabolite repression |
| R | Repression |
| catabolite repression | <biochemistry, molecular biology> Inducible enzyme systems in some microorganisms (such as the lac operon) that are repressed when a more favoured carbon source, such as glucose, is available. Repression in E. Coli is partially relieved if cAMP is bound to the cAMP catabolite activator protein (cAMP receptor protein, CRP) that binds to DNA upstream of the repressed operon concerned. Catabolite repression (of the respiratory system) is seen in yeast in high glucose concentrations, though the mechanism is different. (16 Mar 1998) |
|---|---|
| repression | The inhibition of a gene's expression, this is typically caused by the change in the activity of a regulatory protein. (09 Oct 1997) |
| repression-sensitization | Defense mechanisms involving approach and avoidance responses to threatening stimuli. The sensitizing process involves intellectualization in approaching or controlling the stimulus whereas repression involves unconscious denial in avoiding the stimulus. (12 Dec 1998) |
| primal repression | Repression of material never in conscious thought. (05 Mar 2000) |
| end product repression | Catabolite repression in which the catabolite is an end product of a particular pathway. (05 Mar 2000) |
| enzyme repression | The interference in synthesis of an enzyme due to the elevated level of an effector substance, usually a metabolite, whose presence would cause depression of the gene responsible for enzyme synthesis. (12 Dec 1998) |
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