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¿µ¹® iron deficiency anemia ÇÑ±Û Ã¶°áÇ̺óÇ÷
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  • ¿µ¹®
    ÇѱÛ
  • IgA nephropathy
    IgAÄáÆÏº´(Áõ), IgA½ÅÀ庴(Áõ)
  • acquired immune deficiency syndrome
    ÈÄõ¸é¿ª°áÇÌÁõÈıº, ¿¡ÀÌÁî
  • biotin deficiency syndrome
    ºñ¿Àƾ°áÇÌÁõÈıº
  • deficiency
    1. °áÇÌ 2. °áÇÌÁõ
  • deficiency disease
    °áÇ̺´
  • erythropoietin deficiency anemia
    ¿¡¸®Æ®·ÎÆ÷ÀÌ¿¡Æ¾°áÇ̺óÇ÷
  • fat deficiency disease
    Áö¹æ°áÇ̺´
  • folate deficiency anemia
    ¿±»ê°áÇ̺óÇ÷
  • iron deficiency anemia
    ö°áÇ̺óÇ÷
  • immune deficiency
    ¸é¿ª°áÇÌ
  • leukocyte adhesion deficiency
    ¹éÇ÷±¸ºÎÂø°áÇÌ
  • nutritional deficiency disease
    ¿µ¾ç°áÇ̺´
  • nutritional deficiency state
    ¿µ¾ç°áÇÌ»óÅÂ
  • nutritional deficiency syndrome
    ¿µ¾ç°áÇÌÁõÈıº
  • vitamin deficiency
    ºñŸ¹Î°áÇÌ(Áõ)
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 3 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • IgA nephropathy
    ¸é¿ª±Û·Îºí¸°¿¡ÀÌÄáÆÏº´Áõ, ¸é¿ª±Û·Îºí¸°¿¡À̽ÅÀ庴Áõ
  • iron deficiency anemia
    ö°áÇ̺óÇ÷
  • deficiency
    °áÇÌ(Áõ)
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • acid lipase deficiency
    »ê¼ºÁöÁúºÐÇØÈ¿¼Ò°áÇÌ
  • acquired immune deficiency
    ÈÄõ¸é¿ª°áÇÌ
  • acquired immune deficiency syndrome
    ÈÄõ¸é¿ª°áÇÌÁõÈıº, ¿¡ÀÌÁî
  • adhesion deficiency disorder
    À¯Âø°áÇÌÀå¾Ö
  • antibody deficiency syndrome
    Ç×ü°áÇÌÁõÈıº
  • erythropoietin deficiency anemia
    ¿¡¸®Æ®·ÎÆ÷¿¡Æ¾°áÇ̺óÇ÷
  • folate deficiency anemia
    ¿±»ê°áÇÌ
  • iron deficiency anemia
    ö°áÇ̺óÇ÷
  • ceruloplasmin deficiency
    ¼¼·ê·ÎÇö󽺹ΰáÇÌ
  • complement deficiency
    µµ¿òü°áÇÌ
  • deficiency
    °áÇÌ(Áõ)
  • deficiency disease
    ¿µ¾ç°áÇ̺´
  • fat deficiency disease
    Áö¹æ°áÇÌÁõ
  • functional deficiency
    ±â´É°áÇÌ
  • histogenetic deficiency
    Á¶Á÷¹ß»ý°áÇÌ
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  • ¿µ¹®
    ÇѱÛ
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
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  • ¿µ¹®
    ÇѱÛ
  • IgA, secretory
    ºÐºñÇü IgA
  • anti-IgA
    Ç× IgA Ç×ü
  • iga nephroathy
    ¸é¿ª±Û·ÎºÒ¸° A ½Åº´Áõ
  • immunoglobulin a(iga)
    ¸é¿ª±Û·ÎºÒ¸° A(Øó湡­)
  • Acquiered immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª °áÇÌÁõÈıº
  • Fluorine deficiency
    ºÒ¼Ò°áÇÌÁõ(Ý×áÈÌÀù¹ñø)
  • Folate deficiency
    ¿±»ê°áÇÌÁõ(ç¨ß«ÌÀù¹ñø)
  • Iodine deficiency
    ¿äµå °áÇÌÁõ(ÌÀù¹ñø)
  • Iron deficiency anemia
    ö°áÇ̼ººóÇ÷(ôÑÌÀù¹àõÞ¸úì)
  • Rh deficiency syndrome
    Rh °áÇÌÁõÈıº
  • T cell deficiency
    T¼¼Æ÷°áÇÌ
  • abdominal muscle deficiency syndrome
    º¹±Ù°á¼ÕÁõÈıº(ÜÙÐÉÌÀáßñøý¦ÏØ).
  • acid lipase deficiency
    »ê¼º ¸®ÆÄÁ¦ °áÇÌÁõ(ß«àõ¡­ÌÀù¹ñø)
  • acquired immune deficiency
    ÈÄõ¼º ¸é¿ª°áÇÌ
  • acquired immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª°áÇÌ ÁõÈıº
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • isolated iga deficiency
    ´Üµ¶¼º¸é¿ª±Û·ÎºÒ¸°A°áÇÌ(Ó¤Ô¼àõ Øóæ¹ ¡­ ÌÀù¹)
  • anti-IgA
    Ç× IgA Ç×ü
  • bullous dermatosis,linear iga
    ¼±»ó IgA (àÊß¾¡­)
  • iga nephroathy
    ¸é¿ª±Û·ÎºÒ¸° A ½Åº´Áõ
  • immunoglobulin a(iga)
    ¸é¿ª±Û·ÎºÒ¸° A(Øó湡­)
  • intra-epidermal neutrophilic IgA dermatosis
    Ç¥Çdz»È£Áß±¸¼º ¸é¿ª±Û·ÎºÒ¸°A ÇǺκ´
  • linear IgA bullous dematosis
    ¼±»ó ¸é¿ª±Û·ÎºÒ¸°A ¼öÆ÷¼ºÇǺκ´
  • linear IgA dermatitis
    ¼±»ó ¸é¿ª±Û·ÎºÒ¸°A ÇǺο°
  • linear IgA dermatosis
    ¼±»ó ¸é¿ª±Û·ÎºÒ¸°A ÇǺκ´(Áõ)
  • secretory IgA
    ºÐºñÇü IgA.
  • abdominal muscle deficiency syndrome
    º¹±Ù°á¼ÕÁõÈıº(ÜÙÐÉÌÀáßñøý¦ÏØ).
  • acid lipase deficiency
    »ê¼º ¸®ÆÄÁ¦ °áÇÌÁõ(ß«àõ¡­ÌÀù¹ñø)
  • acquired immune deficiency
    ÈÄõ¼º ¸é¿ª°áÇÌ
  • acquired immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª°áÇÌ ÁõÈıº
  • acquired immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª°áÇÌÁõÈıº(ý­ô¸àõ Øóæ¹ÌÀù¹ñøý¦ÏØ)
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • Deficiency (Nanismus)
    °áÇÌ(³­ÀåÀÌÁõ)
    [¿¾ ¿ë¾î] °áÇÌ
  • Organogenetic deficiency
    ±â°ü¹ß»ý°áÇÌ
    [¿¾ ¿ë¾î] ±â°ü¹ß»ý°áÇÌ
  • Functional deficiency
    ±â´É°áÇÌ
    [¿¾ ¿ë¾î] ±â´ÉÀû°áÇÌ
  • Pituitary hormone deficiency (Pituitary dwarfism)
    ³úÇϼöüȣ¸£¸ó°áÇÌ (³úÇϼöü³­ÀåÀÌ)
    [¿¾ ¿ë¾î] ³úÇϼöüȣ¸£¸ó°áÇÌ (³úÇϼöü³­ÀåÀÌ)
  • Reaction deficiency
    ¹ÝÀÀ°áÇÌ
    [¿¾ ¿ë¾î] ¹ÝÀÀ°áÇÌ
  • Vitamin deficiency (Fetal osteodystrophy)
    ºñŸ¹Î°áÇÌ(žƻÀ¿µ¾çÀå¾Ö)
    [¿¾ ¿ë¾î] ºñŸ¹Î°áÇÌ(žƻÀ¿µ¾çÀå¾Ö)
  • Cellular deficiency (Acallosal cerebrum)
    ¼¼Æ÷°áÇÌ (³úµéº¸°á¿©³ú)
    [¿¾ ¿ë¾î] ¼¼Æ÷°áÇÌ
  • Intracellular deficiency (Albinism)
    ¼¼Æ÷¼Ó°áÇÌ (¹é»öÁõ)
    [¿¾ ¿ë¾î] ¼¼Æ÷³»°áÇÌ
  • Chromosomal deficiency
    ¿°»öü°áÇÌ
    [¿¾ ¿ë¾î] ¿°»öü°á½Ç
  • Stimulation deficiency
    ÀڱذáÇÌ
    [¿¾ ¿ë¾î] ÀڱذáÇÌ
  • Sensory deficiency
    °¨°¢°áÇÌ
    [¿¾ ¿ë¾î] °¨°¢°áÇÌ
  • Thyroid hormone deficiency (Cretinism)
    °©»ó»ùÈ£¸£¸ó°áÇÌ (°©»ó»ù³­ÀåÀÌ)
    [¿¾ ¿ë¾î] °©»ó¼±È£¸£¸ó°áÇÌ (°©»ó»ù³­ÀåÀÌ)
  • Deficiency
    °áÇÌ
    [¿¾ ¿ë¾î] °áÇÌ
  • Deficiency (Monstrous tumor)
    °áÇÌ (±«¹°Á¾)
    [¿¾ ¿ë¾î] °áÇÌ
  • Secretion deficiency
    ºÐºñ°áÇÌ
    [¿¾ ¿ë¾î] ºÐºñ°áÇÌ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 13 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • IgA
    (å²) immunoglobulin A
  • conditioned vitamin deficiency
    Á¶°ÇºÎ(ðÉËìݾ) ºñŸ¹Î°áÇË(ÌÀù¹)
  • deficiency
    °áÇÌ(ÌÀù¹)
  • deficiency disease
    °áÇÌ Áúȯ(ÌÀù¹òðü´)
  • deficiency mutant
    "°áÇÌ º¯ÀÌü(ÌÀù¹Ü¨ì¶ô÷), (ÔÒ) auxotroph"
  • dietary deficiency
    ½ÄÀ̰áÇÌ(ç½å×ÌÀù¹)
  • familial high-density lipoprotein deficiency °¡Á·¼º °í¹Ðµµ ÁöÁú´Ü¹éÁú °áÇÌ (Ê«ðéàõÍÔÚËÓøò·òõ
    Ó±ÛÜòõÌÀù¹)
  • familial lysosomal lipase deficiency
    °¡Á·¼º(Ê«ðéàõ) ¶óÀÌ¼Ò¼Ø ¶óÀÌÆäÀ̽º °áÇÌ(ÌÀù¹)
  • glucose-6-phosphate dehydrogenase deficiency
    ±Û·çÄÚ½º-6-Àλê(×òß«) µðÇÏÀ̵å·ÎÀú³×À̽º °áÇÌ(ÌÀ
  • primary deficiency
    ¿ø¹ß¼º °áÇÌ(ê«Û¡àõÌÀù¹)
  • secondary deficiency
    ÀÌÂ÷ °áÇÌ(ì£ó­ÌÀù¹)
  • simian acquired immune deficiency syndrome
    ¿ø¼þÀÌ ÈÄõ¼º¸é¿ª°áÇÌÁúȯ(ý­ô¸àõØóæ¹ÌÀù¹òðü´)
  • sulfite oxidase deficiency
    ¾ÆÈ²»ê(ä¬üÜß«) ¿Á½Ãµ¥À̽º °áÇÌ(ÌÀù¹)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 6 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • deficiency
    °áÇÌÁõ
  • deficiency anemia
    °áÇ̼ººóÇ÷
  • iron deficiency anemia
    ö°áÇ̼ººóÇ÷
  • mineral deficiency
    ¹«±âÁú°áÇÌ(Áõ)
  • nutritional deficiency disease
    ¿µ¾ç°áÇÌÁõ
  • vitamin deficiency
    ºñŸ¹Î°áÇÌ(Áõ)
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
MD Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major...
Ig Immuno-globulin
  IgA; Immuno-globulin A; (27)(30)60(100) - (170)(80)(250)380 mg/dL
IGA infantile genetic agranulocytosis
IgA immunoglobulin A
S-IgA secretory immunoglobulin A
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
IgA-D IgA deficiency
IgA GN IgA glomerulonephritis
IgA-IC IgA immune complex
IgA NP IgA nephropathy
IgA RF IgA rheumatoid factor
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • iga nephroathy
    ¸é¿ª ±Û·ÎºÒ¸° A ½Åº´Áõ
  • linear IgA disease
    ¼±»ó ¸é¿ª ±Û·ÎºÒ¸° A Áúȯ
  • secretory IgA
    ºÐºñÇü ¸é¿ª ±Û·ÎºÎ¸° A
    Ç÷û, Ÿ¾×, ´«¹°, È£Èí±â, ¼ÒÈ­°ü µîÀÇ ºÐºñ¾× Áß ÇÔÀ¯µÇ¾î ÀÖ´Ù.
  • 17-hydroxylase deficiency
    17-hydroxylase °áÇÌ
  • acquired immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª °áÇÌ ÁõÈıº
    1. ÇöÀúÇÑ ¸é¿ª °áÇ̰ú ÇÔ²² ±âȸ°¨¿°, ¼Ó¹ß¼º ¾Ï ¹× ½Å°æ°è Áõ¼¼°¡ µ¿¹Ý. ¹ÙÀÌ·¯½º ÀÚü¿¡ ÀÇÇÑ º´º¯°ú ¸é¿ª´É·Â ÀúÇÏ¿¡ µû¸¥ ±âȸ °¨¿° µîÀÇ ÀÌÂ÷Àû º´º¯ÀÇ µÎ °¡Áö·Î ´ëº°. HIV¿¡ ÀÇÇØ ¹ß»ýµÇ´Â ÁúȯÀ¸·Î ½Å°æ°è°¡ Áß¿ä Ç¥ÀûÁß Çϳª. ¹ÙÀÌ·¯½º¿¡ °¨¿°µÈ »ç¶÷ÀÇ 40% Á¤µµ°¡ Áúº´ÀÌ ¹ß»ý. ¹ÙÀÌ·¯½ºÀÇ Á÷Á¢ÀûÀÎ ¿µÇâ¿¡ ÀÌÇÑ º´º¯À¸·Î´Â ¸²ÇÁ±¸¼º ¼ö¸·¿°°ú HIV ³ú¿° µîÀÌ ÀÖÀ½. 2. ÈÄõ¼º ¸é¿ª°áÇÌÁõ. Àΰ£ ¸é¿ª°áÇÌ ¹ÙÀÌ·¯½º
  • acquired immune deficiency syndrome
    ÈÄõ¼º ¸é¿ª°áÇÌ ÁõÈıº
  • adenosine deficiency
    ¾Æµ¥³ë½Å °áÇÌÁõ
  • ascorbic acid deficiency
    ¾Æ½ºÄÚ¸£ºó»ê °áÇÌÁõ
    Ư¡ÀûÀÎ ±«Ç÷º´ÀÌ ³ªÅ¸³ª¸ç ÀÌÀÇ Áõ»óÀ¸·Î´Â ÀÕ¸öÀÌ º×°í ½±°Ô ÃâÇ÷ÀÌ µÇ°í, Ä¡¾Æ Çü¼º Àå¾Ö Ä¡Á¶°ñ Èí¼ö ÇÇÇÏ ÃâÇ÷ µîÀÌ ÀÖÀ¸¸ç â»ó Ä¡À¯°¡ ´Ê¾îÁø´Ù.
  • cell adhesion molecular deficiency
    ¼¼Æ÷ À¯Âø ºÐÀÚ °áÇÌ
  • cellular deficiency
    ¼¼Æ÷ °áÇÌ
    ¼¼Æ÷°¡ À¯ÀüÀû ȤÀº ÀÚ°¡¸é¿ªÀû ¿äÀÎÀ¸·Î ÀÎÇØ¼­ °áÇÌµÈ °Í.
  • chromosomal deficiency
    ¿°»öü °áÇÌ
  • color deficiency
    »ö °áÇÌ
  • deficiency anemia
    °áÇ̼º ºóÇ÷
    Ç÷»ö¼Ò °áÇÌÀ¸·Î ÀÎÇÑ ºóÇ÷.
  • deficiency symptom
    °áÇÌ Áõ»ó
    ³»ºÐºñ¼±ÀÇ ºÐºñ Àå¾Ö¿¡ ÀÇÇÑ Áõ»ó.
  • diphosphatase deficiency
    µðÆ÷½ºÆÄŸÁ¦ °áÇÌ
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
IgA deficiency A dysgammaglobulinaemia characterised by a deficiency of IgA.
(12 Dec 1998)
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
glomerulonephritis, IgA Chronic form of glomerulonephritis characterised by recurring haematuria with only slight proteinuria and by deposits of IgA immunoglobulin in the mesangial areas of the renal glomeruli. It usually occurs in young males.
(12 Dec 1998)
secretory component of IgA <immunology, protein> A polypeptide chain of about 60 kD that aids secretion of the IgA, a portion of the IgA receptor on the plasmalemma of the inner side of the epithelial cells lining the gut, which is proteolysed when the IgA receptor complex has travelled through the cell after receptor mediated endocytosis at the inner face, to the outer (luminal) face.
(18 Nov 1997)
IgA <immunology> Major class of immunoglobulin of external secretions in mammals, also found in serum and body fluids such as tears and saliva and in the respiratory, reproductive, urinary and gastrointestinal tracts.
Immunoglobulin A protects the bodys mucosal surfaces from infection and in secretions, it is found as a dimer (400 kD) joined by a short J chain and linked to a secretory portion or transport piece. In serum found as a monomer (170 kD).
Immunoglobulin A is the main mechanism for providing local immunity against infections in the gut or respiratory tract and may act by reducing the binding between an immunoglobulin A coated micro organism and a host epithelial cell. Present in human colostrum but not transferred across the placenta. Have heavy chains.
(30 Mar 1998)
IgA nephropathy <nephrology, pathology> This is a form of glomerulonephritis that results from the deposition of circulating IgA antibody in the kidney tissues.
Inflammation of the glomerulus (glomerulonephritis) is the result. This condition may present as acute glomerulonephritis, chronic glomerulonephritis or rapidly progressive glomerulonephritis.
Berger's is usually detected in an individual with one or two bouts of bloody urine (usually begins during or soon after a respiratory infection) and no other symptoms of renal disease. Only rarely, will Berger's disease permanently affect kidney function and progress to chronic renal failure. This renal disorder more commonly affects males in the 16-40 age group.
Origin: Gr. Pathos = disease
(27 Sep 1997)
IgA, secretory The principle ig found in exocrine secretions such as milk, respiratory and intestinal mucin, saliva and tears. It is composed of two to four molecules of 7s IgA and one molecule each of j chain and secretory component (sc), also called secretory piece. Molecular weight appr. 400,000.
(12 Dec 1998)
IgA-specific serine endopeptidase <enzyme> Extracellular microbial enzymes whose only substrate is human IgA of the iga1 subclass; cleave the immunoglobulin at a specific internal prolyl-threonyl peptide bond in the heavy chain to yield intact faba and fca fragments; consider also EC 3.4.24.13
Registry number: EC 3.4.21.72
Synonym: immunoglobulin a(1) protease, iga1 protease, immunoglobulin a1 protease, IgA protease
(26 Jun 1999)
linear IgA bullous disease in children A rare self-limiting bullous disease, chiefly of the trunk, perioral, and pelvic areas, with onset in the first decade, successively less severe recurrences, and total remission at adolescence; linear epidermal basement membrane zone deposit of IgA is found in involved and in normal skin.
Synonym: linear IgA bullous disease in children.
(05 Mar 2000)
abdominal muscle deficiency syndrome <syndrome> Congenital absence (partial or complete) of abdominal muscles, in which the outline of the intestines is visible through the protruding abdominal wall; in males, genitourinary anomalies (urinary tract dilation and cryptorchidism) are also found; genetics unclear.
(05 Mar 2000)
adult lactase deficiency Onset of lactase deficiency, with resulting milk intolerance and malabsorption, in adulthood. Inherited forms may not be manifested until adulthood; any process that damages the intestinal lining cells can cause lactase deficiency in adults.
(05 Mar 2000)
alpha-1 antitrypsin deficiency <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues.
The lack of this protein leads to damage of various organs, but mainly to the lung and liver.
symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant
(12 Dec 1998)
alpha-1-proteinase deficiency Absence of a serum proteinase inhibitor that may cause nodular non-suppurative panniculitis.
(05 Mar 2000)
alpha-antitrypsin deficiency <enzyme> A specific enzyme (alpha 1 antitrypsinase) that when absent genetically can result in panacinar emphysema (lung disease) and liver disease.
There is no specific treatment for this condition other than supportive care for the liver and lung complications.
Medications such as alpha-1proteinase inhibitor is given regularly to these patients.
Incidence: approximately 1 in 10,000.
(02 Jan 1998)
anaemia, iron deficiency Deficiency of iron results in anaemia because iron is necessary to make haemoglobin, the key molecule in red blood cells responsible for the transport of oxygen. In iron deficiency anaemia, the red cells are unusally small (microcytic) and pale (hypochromic). Characteristic features of iron deficiency anaemia in children include failure to thrive (grow) and increased infections. The treatment of iron deficiency anaemia, whether it be in children or adults, is with iron and iron-containing foods. Food sources of iron include meat, poultry, eggs, vegetables and cereals (especially those fortified with iron). According to the National Academy of Sciences, the Recommended Dietary Allowances of iron are 15 milligrams per day for women and 10 milligrams per day for men.
Anaemia characterised by low or absent iron stores, low serum iron concentration, elevated free erythrocyte porphorin, low transferrin saturation, elevated transferrin, low serum ferritin, low haemoglobin concentration or haematocrit, and hypochromic microcytic red blood cells. Symptoms may include pallor, angular stomatitis and other oral lesions, gastrointestinal complaints, retinal haemorrhages and exudates, and thinning and brittleness of the nails. Among the causes of iron-deficiency anaemia are inadequate iron intake, impaired iron absorption, increased blood loss and increased requirements such as infancy, pregnancy, and lactation.
(12 Dec 1998)
antibody deficiency disease <syndrome> Any of a group of disorders associated with a defective antibody production due to defects in the B-type lymphocyte system or in T-type lymphocytes; chief manifestation is an increased susceptibility to infection by various microorganisms.
See: agammaglobulinaemia, hypogammaglobulinaemia, immunodeficiency.
Synonym: antibody deficiency disease.
(05 Mar 2000)
antibody deficiency syndrome <syndrome> Any of a group of disorders associated with a defective antibody production due to defects in the B-type lymphocyte system or in T-type lymphocytes; chief manifestation is an increased susceptibility to infection by various microorganisms.
See: agammaglobulinaemia, hypogammaglobulinaemia, immunodeficiency.
Synonym: antibody deficiency disease.
(05 Mar 2000)
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